LIANG Xiao, QIAN Haibing. Progress of the Relationship between Aquaporin 4 and Post-Stroke Cognitive Impairment[J]. Chinese Journal of Stroke, 2022, 17(12): 1385-1390.
[1]中国卒中学会血管性认知障碍分会. 卒中后认知障碍管理专家共识2021[J]. 中国卒中杂志,2021,16(4):376-389.
[2]VANDEBROEK A,YASUI M. Regulation of AQP4 in the central nervous system[J/OL]. Int J Mol Sci,2020,21(5):1603[2021-11-01]. https://doi.org/10.3390%2Fijms21051603.
[3]WANG Y F,HUANG C Y,GUO Q H,et al. Aquaporin-4 and cognitive disorders[J]. Aging Dis,2022,13(1):61-72.
[4]VENKAT P,CHOPP M,ZACHAREK A,et al. White matter damage and glymphatic dysfunction in a model of vascular dementia in rats with no prior vascular pathologies[J/OL]. Neurobiol Aging,2017,50:96-106[2021-11-01]. https://doi.org/10.1016/j.neurobiolaging.2016.11.002.
[5]DENVER P,D'ADAMO H,HU S X,et al. A novel model of mixed vascular dementia incorporating hypertension in a rat model of Alzheimer's disease[J/OL]. Front Physiol,2019,10:1269[2021-11-01]. https://doi.org/10.3389/fphys.2019.01269.
[6]HASE Y,CRAGGS L,HASE M,et al. Effects of environmental enrichment on white matter glial responses in a mouse model of chronic cerebral hypoperfusion[J/OL]. J Neuroinflammation,2017,14(1):81[2021-11-01]. https://doi.org/10.1186/s12974-017-0850-5.
[7]REED M M,BLAZER-YOST B. Changes in TRPV4,AQP1,and AQP4 in a genetic model of hydrocephalus[J/OL]. FASEB J,2022,36(S1):R4957[2021-11-01]. https://doi.org/10.1096/fasebj.2022.36.S1.R4957.
[8]KITCHEN P,SALMAN M M,HALSEY A M,et al. Targeting aquaporin-4 subcellular localization to treat central nervous system edema[J]. Cell,2020,181(4):784-799.
[9]HUBBARD J A,SZU J I,BINDER D K,et al. The role of aquaporin-4 in synaptic plasticity,memory and disease[J/OL]. Brain Res Bull,2018,136:118-129[2021-11-01]. https://doi.org/10.1016/j.brainresbull.2017.02.011.
[10]ILIFF J J,CHEN M J,PLOG B A,et al. Impairment of glymphatic pathway function promotes tau pathology after traumatic brain injury[J]. J Neurosci,2014,34(49):16180-16193.
[11]SZU J I,BINDER D K. The role of astrocytic aquaporin-4 in synaptic plasticity and learning and memory[J/OL]. Fron Integr Neurosci,2016,10:8[2021-11-01]. https://doi.org/10.3389/fnint.2016.00008.
[12]ZEPPENFELD D M,SIMON M,HASWELL J D,et al. Association of perivascular localization of aquaporin-4 with cognition and Alzheimer disease in aging brains[J]. JAMA Neurol,2017,74(1):91-99.
[13]ROSU G C,CATALIN B,BALSEANU T A,et al. Inhibition of aquaporin 4 decreases amyloid Aβ40 drainage around cerebral vessels[J]. Mol Neurobiol,2020,57(11):4720-4734.
[14]IKESHIMA-KATAOKA H,ABE Y,ABE T,et al. Immunological function of aquaporin-4 in stab-wounded mouse brain in concert with a pro-inflammatory cytokine inducer,osteopontin[J/OL]. Mol Cell Neurosci,2013,56:65-75[2021-11-01]. https://doi.org/10.1016/j.mcn.2013.02.002.
[15]GENEL O,PARIANTE C M,BORSINI A. The role of AQP4 in the pathogenesis of depression,and possible related mechanisms[J/OL]. Brain Behav Immun,2021,98:366-377[2021-11-01]. https://doi.org/10.1016/j.bbi.2021.08.232.
[16]LU G Y,PANG C,CHEN Y,et al. Aquaporin 4 is involved in chronic pain but not acute pain[J/OL]. Behav Brain Res,2020,393:112810[2021-11-01]. https://doi.org/10.1016/j.bbr.2020.112810.
[17]卢鹤扬,冯一苇,崔梅. 类淋巴系统与中枢神经系统病变的相关性研究进展[J]. 中国临床神经科学,2020,28(1):78-84,92.
[18]易婷,金硕果,尹海燕,等. 脑胶质淋巴系统—脑卒中病理机制的新探索[J]. 中国全科医学,2022,25(9):1136-1140.
[19]KIMURA N,TAKAHASHI M,TASHIRO T,et al. Amyloid beta up-regulates brain-derived neurotrophic factor production from astrocytes:rescue from amyloid beta-related neuritic degeneration[J]. J Neurosci Res,2006,84(4):782-789.
[20]GRIGOLASHVILI M A,MUSTAFINA R M. The role of the inflammatory process in the development of post-stroke cognitive impairments[J/OL]. Zh Nevrol Psikhiatr Im S S Korsakova,2021,121(3. Vyp. 2):16-21[2021-11-01]. https://doi.org/10.17116/jnevro202112103216.
[21]SINGH D. Astrocytic and microglial cells as the modulators of neuroinflammation in Alzheimer’s disease[J/OL]. J Neuroinflammation,2022,19(1):206[2021-11-01]. https://doi.org/10.1186/s12974-022-02565-0.
[22]HUANG K L,HSIAO I T,HO M Y,et al. Investigation of reactive astrogliosis effect on post-stroke cognitive impairment[J/OL]. J Neuroinflammation,2020,17(1):308[2021-11-01]. https://doi.org/10.1186/s12974-020-01985-0.
[23]卢丽敏,智孔亮. 基质金属蛋白酶9对缺血性脑卒中患者认知功能障碍的影响[J]. 中国临床医生杂志,2021,49(2):179-182.
[24]贺伟,展宏刚. SIRT1对早期脑缺血大鼠水通道蛋白4表达的影响[J]. 中国病理生理杂志,2017,33(3):455-461.
[25]宋轶群,刘梅,于广. 丁苯酞对急性脑梗死患者认知功能和血清学指标的影响及二者相关性分析[J]. 中国药房,2017,28(2):243-246.
[26]陈艳红,陈丽芳,张小梅,等. 丁苯酞对脑梗死后脑水肿患者血清Hcy、AQP4、MMP-9水平和神经功能的影响[J]. 中国医学创新,2022,19(28):32-36.
[27]LI L H,ZHANG H,VARRIN-DOYER M,et al. Proinflammatory role of aquaporin-4 in autoimmune neuroinflammation[J]. FASEB J,2011,25(5):1556-1566.
[28]HIRT L,FUKUDA A M,AMBADIPUDI K,et al. Improved long-term outcome after transient cerebral ischemia in aquaporin-4 knockout mice[J]. J Cereb Blood Flow Metab,2017,37(1):277-290.
[29]陈红梅,秦碧勇,尤志珺. 丁苯酞治疗急性缺血性脑卒中患者的疗效评价[J]. 湖北医药学院学报,2020,39(1):52-56.
[30]CORREIA S C,PERRY G,MOREIRA P I. Mitochondrial traffic Jams in Alzheimer's disease-pinpointing the roadblocks[J]. Biochim Biophys Acta,2016,1862(10):1909-1917.
[31]LI Y K,WANG F,WANG W,et al. Aquaporin-4 deficiency impairs synaptic plasticity and associative fear memory in the lateral amygdala:involvement of downregulation of glutamate transporter-1 expression[J]. Neuropsychopharmacology,2012,37(8):1867-1878.
[32]LAN Y L,ZOU S,CHEN J J,et al. The Neuroprotective effect of the association of aquaporin-4/glutamate transporter-1 against Alzheimer's disease[J/OL]. Neural plast,2016:4626593[2021-11-01]. https://doi.org/10.1155/2016/4626593.
[33]YAN P Y,XUE Z C,LI D Z,et al. Dysregulated CRTC1-BDNF sig‐naling pathway in the hippocampus contributes to Aβ oligomer-induced long-term synaptic plasticity and memo‐ry impairment[J/OL]. Exp Neurol,2021,345:113812[2021-11-01]. https://doi.org/10.1016/j.expneurol.2021.113812.
[34]李成武. AQP4调节星形胶质细胞分泌BDNF的机制研究[D]. 南京:南京中医药大学,2019.
[35]SKUCAS V A,MATHEWS I B,YANG J M,et al. Impairment of select forms of spatial memory and neurotrophin-dependent synaptic plasticity by deletion of glial aquaporin-4[J]. J Neurosci,2011,31(17):6392-6397.
[36]PLUTA R. Astroglial expression of the beta-amyloid in ischemia-reperfusion brain injury[J/OL]. Ann N Y Acad Sci,2002,977:102-108[2021-11-01]. https://doi.org/10.1111/j.1749-6632.2002.tb04803.x.
[37]陈慧杰,孙东东. 血清S-100β神经元特异性烯醇化酶水平与脑卒中患者神经功能损害程度的相关性研究[J]. 中国实用神经疾病杂志,2021,24(5):420-424.
[38]TARASOFF-CONWAY J M,CARARE R O,OSORIO R S,et al. Clearance systems in the brain-implications for Alzheimer disease[J]. Nat Rev Neurol,2015,11(8):457-470.
[39]LI G,HE X F,LI H,et al. Overexpression of SLIT2 improves function of the paravascular pathway in the aging mouse brain[J]. Int J Mol Med,2018,42(4):1935-1944.
[40]XU Z Q,XIAO N,CHEN Y L,et al. Deletion of aquaporin-4 in APP/PS1 mice exacerbates brain Aβ accumulation and memory deficits[J/OL]. Mol Neurodegener,2015,10(1):58[2021-11-01]. https://doi.org/10.1186/s13024-015-0056-1.
[41]HE X F,LIU D X,ZHANG Q,et al. Voluntary exercise promotes glymphatic clearance of amyloid beta and reduces the activation of astrocytes and microglia in aged mice[J/OL]. Fron Mol Neurosci,2017,10:144[2021-11-01]. https://doi.org/10.3389/fnmol.2017.00144.
[42]WANG F R,HUA S Y,ZHANG Y,et al. Associationbetween small vessel disease markers,medial temporallobe atrophy and cognitive impairment after stroke:a systematic review and meta-analysis[J/OL]. J Stroke Cerebrovasc Dis,2021,30(1):105460[2021-11-01]. https://doi.org/10.1016/j.jstrokecerebrovasdis.2020.105460.
[43]王珵,陶汉川,姜小兰,等. 缺血性脑卒中后认知功能障碍与梗死部位及体积的关系分析[J]. 吉林医学,2021,42(7):1626-1631.
[44]YAO X M,DERUGIN N,MANLEY G T,et al. Reduced brain edema and infarct volume in aquaporin-4 deficient mice after transient focal cerebral ischemia[J/OL]. Neurosci Lett,2015,584:368-372[2021-11-01]. https://doi.org/10.1016/j.neulet.2014.10.040.
[45]LI YAN,LIU S L,QI S H. ALDH2 protects against ischemic stroke in rats by facilitating 4-HNE clearance and AQP4 down-regulation[J]. Neurochem Res,2018,43(7):1339-1347.
[46]石向群,杨金升,王运良,等. 水通道蛋白4基因干预对大鼠脑缺血再灌注损伤后梗死灶体积的影响[J]. 中华老年医学杂志,2005,24(3):201-204.