›› 2009, Vol. 4 ›› Issue (01): 67-73.

• 综述 • 上一篇    下一篇

低密度脂蛋白与卒中

王艺铮,王拥军   

  1. 100050 北京市 首都医科大学附属北京天坛医院神经内科
  • 收稿日期:2008-10-06 修回日期:1900-01-01 出版日期:2009-01-20 发布日期:2009-01-20
  • 通讯作者: 王拥军

Low Density Lipoprotein and Stroke

  • Received:2008-10-06 Revised:1900-01-01 Online:2009-01-20 Published:2009-01-20

摘要: 降脂治疗可降低卒中的发病率,正确的血脂基线水平的测定对降脂方案的确定有重要意义。有研究发现,卒中急性期血脂水平存在变化,给评价患者的血脂水平带来一定的困难。本文将对低密度脂蛋白导致卒中的发病机制以及卒中急性期低密度脂蛋白的变化及其相关内容进行综述。

关键词: 卒中; 急性期; 胆固醇; LDL

Abstract: Clinical trials have shown that cholesterol-lowering therapy can reduce the incidence of stroke, so a correct lipids assessment of baseline is important to establish cholesterol-lowering strategy. But some researches have found that lipid levels are variable after acute stroke, which makes it difficult to assess lipid level. This article reviews the pathogenic mechanism of low density lipoprotein-cholesterol(LDL-C) inducing stroke, changes of LDL-C after acute stroke and other related contents.

Key words: Stroke; acute; Cholesterol; LDL