›› 2010, Vol. 5 ›› Issue (10): 852-856.

• 综述 • 上一篇    下一篇

卒中后抑郁的发病机制研究进展

侯晶晶1,张宁2,陈立云2,王春雪2   

  1. 1 北京市北京市第六医院神经内科(首都医科大学2009级同等学力硕士研究生)2首都医科大学附属北京天坛医院神经内科
  • 收稿日期:2010-05-20 修回日期:2010-04-20 出版日期:2010-10-20 发布日期:2010-10-20
  • 通讯作者: 王春雪

Advances in Research on Pathogenesis for Patients with Post-Stroke Depression

HOU Jing-Jing*, ZHANG Ning, CHEN Li-Yun, et al.   

  • Received:2010-05-20 Revised:2010-04-20 Online:2010-10-20 Published:2010-10-20
  • Contact: WAGN Chun-Xue

摘要: 卒中后抑郁(post-stroke depression,PSD)是卒中的常见并发症。PSD的发病机制复杂,涉及神经生物学、解剖学、社会学和心理学等诸多因素。本文对以神经递质学及神经解剖学改变为代表的机制学说的研究进展做一综述,以期加深对PSD的理解和认识。

关键词: 抑郁; 卒中后; 发病机制

Abstract: Post-stroke depression (PSD) is a common complication of cerebrovascular disease.The pathogenesis of PSD is complex, which refers to many factors just like neurobiology, anatomy,sociology and psychology. This article reviews the advances in the research on pathogenesis ofneurotransmitters and neuroanatomy, so as to strengthen understanding and recognition of PSD.

Key words: Depression; post-stroke; Pathogenesis